Economic, sociological, natural, anthropogenic constructed systems and associated ambients have been enmeshed in the untoward impacts of the COVID-19 pandemic. Environment, health and quality of life should remain of immediate concern in governmental policy and governance. In this COVID-19 era, the diminished introduction of pollutants and the decreased exacerbation of contaminants in the ecosystem due to drastic reduction in economic and industrial production have perspicuously posed less threat and aberration to biodiversity, ecological and environmental systems. The conceptual ecological analysis of the COVID-19 trajectories exposes the dire consequences of the unpreparedness of nature and humans in this pandemic era. It is important to elucidate the functionalities of anthropogenic activities in environmental degradation and the inextricable-linkage between pandemics, ecosystem deterioration and health as pertinent to animal, human, plant, land usage, water and biodiversity. The conspiracy theories, ecological fallacy and mass hysteria of COVID-19 emergence stem from perspicuous extensive shirking of responsibilty regarding domestic and global environmental health by the public and private sectors of production, consumption, administration and governance.

Drought stress directly affects the growth along with productivity of plants by altering plant water status. The present study was carried out to characterize the genetic variability for seedling and morpho-physiological parameters in different sunflower genotypes under water stressed conditions. A total twenty seven genotypes including two hybrids, eight advanced lines and seventeen accessions of sunflower (Helianthus annuus L.) were tested against drought stress at seedling stages by applying Polyethylene glycol (PEG). Significant means were calculated among traits using analysis of variance (ANOVA) whereas, correlation and principle component analysis also confirmed that germination percentage, root length, shoot length, chlorophyll content, stomatal frequency are positively linked with each other hence, these traits were responsible for most of variation among genotypes. The cluster analysis showed that genotypes Ausun, line-1, line-2, line-3, 17578, 17570, line-6, line-7 and 17562 are more diverse among all the genotypes.

Medicinal plants contain substances with high therapeutic value because they contain multiple bioactive chemicals. Chemical analysis of Indigofera tinctoria leaves, stem bark and roots was evaluated. The result revealed that proximate composition of Indigofera tinctoria leaves contained 12.49 % moisture content (M.C), 87.51 % dry matter (DM), 30.53 % crude protein (CP), 19.02 % crude fibre (CF), 2.44 % ether extract (EE), 13.86 % ash, 36.59 % carbohydrate (CHO) and 254.1 kcal/100g energy (ME). Indigofera tinctoria stem bark contained M.C (6.40 %), DM (93.60 %), CP (5.11 %), CF (54.49 %), EE (2.00 %), ash (10.42 %), CHO (29.98 %) and ME (156.0 kcal/100g). Indigofera tinctoria roots contained MC, DM, CP, CF, EE, ash, CHO and ME at 10.04 %, 89.60 %, 8.22 %, 40.88 %, 1.21 %, 8.43 %, 42.47 % and 210.0 kcal/100g respectively. Vitamin analysis showed that Indigofera tinctoria leaves, stem bark and roots contained ß- carotene (8.45, 2.88 and 5.11 mg/100 g), Vitamin B1 (1.94, 0.33 and 1.00 mg/100 g), Vitamin B2 (0.71, 0.21 and 0.50 mg/100 g), Vitamin B3 (0.66, 0.34 and 0.48 mg/100 g), Vitamin B6 (0.32, 0.21 and 0.30 mg/100 g), Vitamin B7 (0.63, 0.01 and 0.16 mg/100 g), Vitamin B9 (0.26, 0.10 and 0.18 mg/100 g), Vitamin B12 (0.21, 0.03 and 0.10 mg/100 g), Vitamin C (14.0, 3.56 and 9.44 mg/100 g), Vitamin D (0.10, 0.01 and 0.06 mg/100 g) and Vitamin K (0.17, 0.07 and 0.12 mg/100 g). Amino acid analysis revealed the presence of threonine, leucine, lysine, valine, tryptophan, glycine, phenylalanine, histidine, methionine, alanine, serine, proline, aspartate, glutamic acid, tryrosine and cysteine in Indigofera tinctoria leaves, stem bark and roots at (7.65 %, 1.22 % and 3.03 %), (5.76, 1.09 % and 2.46 %), (3.11 %, 1.21% and 2.00 %), (7.21 %, 3.53 % and 4.09 %), (1.45%, 0.03% and 1.00 %), (4.76 %, 0.08 % and 2.33 %), (6.33 %, 2.45 % and 3.49 %), (7.42 %, 2.00 % and 3.00 %), (3.49 %, 0.01 % and 2.00 %), (2.41 %, 0.56 % and 1.20 %), (5.23 %, 1.22 % and 1.76 %), (2.87 %, 0.57 % and 1.00 %), (5.32 %, 2.11 % and 3.56 %), (9.66 %, 4.21 % and 5.11 %), (2.45 %, 0.57 % and 1.67 %) and (1.85 %, 0.81 % and 0.89 %) respectively. It was concluded that Indigofera tinctoria leaves, stem bark and roots are loaded with significant quantity of nutrients, vitamins and amino acid (leaves ? roots ? stem bark).

Sandalwood (White Sandal) is the fragrant heartwood of some species of genus Santalum. The widely distributed and economically important Santalum genus belongs to the family Santalaceae which includes 30 genera with about 400 species, many of which being completely or partially parasitic (John, 1947). The word Sandal has been derived from Chandana (Sanskrit), Chandan (Persian), Savtador (Greek) and Santal (French). There are references of Sandalwood in Indian mythology, folklore and ancient scripts. ‘Chandana’ the Sanskrit name ascribed to Santalum album L. was known and used in India from the earliest historic times and is frequently mentioned in the ancient Sanskrit writings, some of which dated before Christian era. Kautilya’s Arthashastra (320 B.C.) considered Sandal as one of the important forest products to increase royal revenue. Charaka Sanhitha, the major text book of internal medicine in Ayurveda (300 B.C.) quotes uses of Sandal over 160 time in the entire text. In treatment of major diseases like fever, piles, hemorrhagic conditions, diabetes, dropsy, mental disorders, management of poisons & skin disorders wide spread uses of sandal is seen. Susrutha Samhita (150 B.C.) a great text on Indian wisdom on surgical procedures, equally preferred sandal for the management of wounds.

Schindler syndrome is an inherited genetic disorder that mainly causes neurological problems. Schindler syndrome is caused by a mutation in the NAGA gene, which is located in the long arm of chromosome 22 as 22q13.2.